US EditionCRISPR/dCas9-TET1–Mediated Epigenetic Editing Reactivates miR-200c in Breast Cancer Cells
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CRISPR/dCas9-TET1–Mediated Epigenetic Editing Reactivates miR-200c in Breast Cancer Cells
Aberrant DNA methylation of tumor-suppressive microRNAs is a frequent epigenetic alteration driving cancer progression. This study explores the targeted demethylation of the miR-200c promoter, a key regulator of epithelial–mesenchymal transition (EMT), using the CRISPR/dCas9-TET1 system in breast...
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