US EditionDual PDE4/10 Inhibition Restores CREB1 Function and Enhances Neuronal Resilience in Alzheimer's Disease
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Dual PDE4/10 Inhibition Restores CREB1 Function and Enhances Neuronal Resilience in Alzheimer's Disease
Background Alzheimer’s disease (AD) involves cognitive decline, amyloid-beta (Aβ) accumulation, tau hyperphosphorylation, and neuroinflammation. CREB1, a key transcription factor for memory, is downregulated in AD, contributing to disease progression. Methods
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