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Astrocytic Mettl14 Depletion Enhances Cognitive Function by Attenuating Astrogliosis via the DUSP1/MAPK Pathway in APP/PS1 Mice: Targeting Neuroinflammation in Alzheimer’s Disease
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Astrocytic Mettl14 Depletion Enhances Cognitive Function by Attenuating Astrogliosis via the DUSP1/MAPK Pathway in APP/PS1 Mice: Targeting Neuroinflammation in Alzheimer’s Disease
Investigators used a Mettl14 conditional knockout APP/PS1 mouse model to investigate the effects of modulating astrocytic N6-methyladenosine levels on Alzheimer’s disease progression. [Molecular Psychiatry] AbstractGraphical Abstract
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