US EditionThis missing vitamin could stop cancer cells in their tracks
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2 Articles
This missing vitamin could stop cancer cells in their tracks
Cancer cells are known for their “glutamine addiction,” but many can escape this weakness by switching to alternative fuels. Researchers found that vitamin B7 acts like a metabolic “license,” enabling this escape route through a key enzyme. Without biotin, cancer cells lose that flexibility and stop growing. Mutations in a cancer-linked gene can make this vulnerability even stronger, offering a promising new target for therapy.
A recent study from the University of Lausanne has identified a new vulnerability in cancer cells related to vitamin B7, also known as biotin. The research highlights how the dependence of tumor cells on glutamine can be exploited to develop new therapeutic strategies. Cancer cell dependence on glutamine: Cancer cells are often characterized by a strong dependence on glutamine, an essential amino acid for protein synthesis and DNA replication. T…
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